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Professor Douglas Kell from the School of Chemistry at Manchester University and the Manchester Institute of Biotechnology is one of the authors of the editorial. He says supposedly sterile red blood cells contain dormant microbes, which also affects blood transfusions.

"We say that there is indisputable evidence that Alzheimer's disease has a dormant microbial component and that it can be woken up by iron dysregulation. Removing this iron will slow or prevent cognitive degeneration - we cannot continue ignore all evidence, "said Professor Douglas Kell.

Professor Resia Pretorius of the University of Pretoria, who worked with Douglas Kell on the editorial, said: "Microbial presence in the blood can also play a fundamental role in causing systemic inflammation, which is a hallmark of Alzheimer's disease - particularly the bacterial cell wall component and the Endotoxin, the lipopolysaccharide. In addition, there are numerous indications that this can cause neuroinflammation and amyloid-β-plaque formation ".

The results of this editorial could also impact future treatment of Parkinson's disease and other progressive neurological disorders.

Microbes and Alzheimer's disease

We are researchers and clinicians studying Alzheimer's disease (AD) or related topics, and we write to express our concern that a certain aspect of the disease has been neglected even though treatment based on it has progressed the AD could slow down or stop. We refer to the many studies, especially in humans, that identify specific microbes in the brain of the elderly, particularly the herpes simplexVirus Include type 1 (HSV1), Chlamydia pneumoniae and various types of spirochetes in the etiology of Alzheimer's disease. A fungal infection of the brain in AD [5, 6] has also been described, as have abnormal microbes in the blood of AD patients. The first observations of HSV1 in the brains of Alzheimer's patients were reported almost three decades ago]. The steadily growing number of these studies (now around 100 on HSV1 alone) justifies a reassessment of the infection and Alzheimer's concept.

AD is associated with neuronal loss and progressive synaptic dysfunction, accompanied by deposition of the amyloid-β (Aβ) peptide, a cleavage product of the amyloid-β protein precursor (AβPP), and abnormal forms of the tau protein, markers that serve as diagnostic criteria were used for the disease. These are the markers of AD, but it is not known whether they are the cause or the consequences of AD. We suspect that these are indicators of an infectious etiology. Alzheimer's disease is often unaware that microbes can cause both chronic and acute diseases, that some microbes can remain latent in the body and have the potential for reactivation, the effects of which can occur years after the initial infection, and that People may be infected but not necessarily affected, so the "controls", even if they are infected, are asymptomatic.

"Microbes and Alzheimer's" by Itzhaki, Ruth F .; Lathe, Richard; Balin, Brian J .; Ball, Melvyn J .; Träger, Elaine L .; Bullido, Maria J.Carter, Chris; Clerici, Mario; Cosby, S. Louise; Field, Hugh; Fulop, Tamas; Grassi, Claudio; Griffin, W. Sue T .; Haas, Jürgen; Hudson, Alan P .; Kamer, Angela R .; Kell, Douglas B.Licastro, Federico; Letenneur, Luc; Lövheim, Hugo; Mancuso, Roberta; Miklossy, Judith; Lagunas, Carola Otth; Palamara, Anna Teresa; Perry, George; Preston, Christopher; Pretorius, Etheresia; Strandberg, Timo; Tabet, Naji; Taylor-Robinson, Simon D .; and Whittum-Hudson, Judith A. in Journal of Alzheimer's Disease. Published online on March 8, 2016 doi: 10.3233 / JAD-160152

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Article author:
Herbert Eder

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